re: LSU Pathologists Reveal Unexpected Heart Damage from Covid-19

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Since none of you actually read the link... one of the first things they are quoted saying is that they want to study it further for potential treatment interventions.

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Yet the title of the article reads

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Autopsies reveal surprising cardiac changes in COVID-19 patients

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Or is it from the mcdonalds they all gorge on?

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The same science that told us 3 months ago that masks weren't necessary


This was a poor attempt to get everyone to stop buying all the masks so they could be used for medical purposes. We knew then it was bull shite.

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Um, yeah. That's my point, as you have pointed out, scientists and medical professionals will withhold the truth or outright lie to fit their agenda.

That's probably actually due to alcohol.

And yet the MSM still won’t entertain the idea that this thing was engineered.

By Gates, Fauci and Obama. Amirite?

let me show you how this works. if you read the actual publication....

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The most significant finding was severe right
ventricular dilation (defined as right: left ventricular cavity diameter ratio of =1:1) in 9 cases
H&E stained sections showed neither large areas of myocyte necrosis, nor significant interstitial
lymphocytic infiltrate (Figure 1B). The sections were remarkable for scattered individual
myocyte necrosis (Figure 1B). Electron microscopy (EM) revealed particles consistent with SARSCoV-2 virus in the myocardial endothelial compartment, pneumocytes, and renal tubular
epithelium, but not in the myocytes of 6 patients examined by EM (Figure 1C, G, H). By
comparison, post-mortem analysis of patients who died from the SARS epidemic confirmed viral
genome in 35% of the hearts examined
there was no pathological
evidence of typical lymphocytic myocarditis. Rather, the hearts showed a pattern of individual
cell dropout/necrosis/apoptosis not associated with any lymphocytic infiltrate

The mechanism of cardiac injury in COVID-19 is not known. Our earlier report [1]
indicates that our patients suffered from diffuse alveolar damage, and pulmonary thrombotic and
microangiopathic changes. Based on this interpretation, and the acute RV dilatation seen in nine
patients, the elevated biomarkers may reflect extreme stress secondary to acute pulmonary
disease
The mechanism of cardiac injury in COVID-19 is not known. Our earlier report [1]
indicates that our patients suffered from diffuse alveolar damage, and pulmonary thrombotic and
microangiopathic changes. Based on this interpretation, and the acute RV dilatation seen in nine
patients, the elevated biomarkers may reflect extreme stress secondary to acute pulmonary
disease. Our EM data showing viral infection of the endothelial compartment is consistent with
a recent paper by Chen et al., which showed that despite representing a low absolute number of
cells in human hearts, pericytes contain up to 16% of the SARS-CoV-2 infected cell population
and may cause capillary endothelial cell/microvascular dysfunction, leading to increased
thrombosis and individual cell necrosis and/or apoptosis [4

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let me summarize.

First, the most significant finding was an enlarged heart chamber due to overwhelming lung inflammation putting strain on the heart. This is not unusual for any serious lung disease.

Next, they did not find covid in heart tissue, they found random heart cells that were damaged and some virus in small blood vessels that could possibly be causing clotting issues.

When you actually read the paper it's not nearly as bad as the headline sounds. They claim its not typical myocarditis (inflammation of the heart) like that is a bad thing, I actually think it is a good thing that there isn't significant inflammation. I don't see anything in this letter that is concerning.

Covid appears to effect the heart less than SARS1

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Although there were other underlying conditions, the majority had high blood pressure, half had insulin-treated type 2 diabetes, and about 41% had obesity.

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So you're telling us that the fact that they had pickled pig feet and lips, cracklins, boudin, and fried chicken as a daily main diet didn't help?

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Actually thought the findings were rather reassuring that this virus isn't adding another way to kill a person. It seems to correlate well with what would be expected from diffuse alveolar damage and its sequela leading to right heart failure and myocardial damage in that respect as opposed to direct cytotoxic effects on the myocardial cells themselves.

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Exactly!

I wish they would have commented on the amount of cellular damage

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The heart damage is more likely from the hardening of arteries due to the high triglycerides in the french fries and fried food these people ate.

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I am assuming that most people who die with these same underlying conditions who do not have covid do not have this same heart damage or they wouldn't be reporting this.