What statin are you on?

Crestor

Get on Repatha. It's not a statin. My numbers did a 180 after a few months.

Just take Crestor. If you can’t tolerate then get on repatha.

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Any side effects of the shot?

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Very few. Except for possibly high blood sugar. Not sure on costs, insurance covers it. 1 injection every two weeks

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Damn I already have borderline fasting glucose at 92.

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What's your A1C?

I just found out it’s not part of my yearly physical blood work

I’m getting a list together of stuff. I’m gonna request for my discount online lab.

I’ll just take the orders down to a quest down the street

Apo B, LP little A, a1c

Sorry I meant to say I had my little A checked and its 27 nmol

Rosuvastatin (=Crestor), 10 mg, no side effects, certainly not joint pain, that I can discern - started treatment a year ago. I’m also a tall, thin guy - healthy - but older than you. Your blood panel lipids though borderline high for LDL are not particularly bad, mine were similar to yours for years and my PCP and cardiologist left it up to me as to whether or not I wanted to take a statin, I declined, as there was no history of heart disease in my family on father’s and mother’s side - and my physicians were good with that.

However, I did go on the statin and low dose aspirin a year ago after a test showed I had a CAC score of 550. As mentioned by another poster, and my cardiologist, it reduced LDL and Total C 40%. Given your family history of heat disease, CAC score and age, and your cardiologists’s advice I wouldn’t discount going back on a statin. My cardiologist did tell me that if one type statin has negative side effects it’s likely another type of statin will likely have similar side effects….so there is that. Maybe dose regulation is key for you to minimize negative side effects.

Never smoked or done drugs. Very casual drinker

Be cautious with statins. More and more evidence is coming out that they are not the panacea once thought.

Statins do reduce cholesterol BUT at what cost? Our bodies produce cholesterol (mainly in the liver) for many vital functions. We would die quickly if not for cholesterol.

Our brains consist of about 20% cholesterol. Is that the reason that 'brain fog' occurs when taking statins? What about dementia? IDK

There is still a lot of debate going on in cardiologist circles about statins.

I picked up this book a few years ago and it and other studies have changed my mind.
The Great Cholesterol Myth

I am no medical professional, and I certainly do not claim to be an expert in this matter. I am just a regular guy trying to keep healthy and learn about health so I can live a good life. Heart disease runs in my family, so I have a huge stake in this.

Folks with normal cholesterol have heart attacks, strokes and embolisms all the time.

LDL's main function is to carry cholesterol from the liver to other tissues in the body. HDL carry's excess cholesterol back to the liver.

The big issue with LDLs is when they become dense and small, they burrow into the thin lining of the arteries, the endothelium, and build up over time like a pimple (soft plaque) and erupt like a volcano causing clots, aka blockage, which results in heart attacks, strokes, and embolisms.

Most of the time, enzymes in our body breaks up those clots and the arteries heal with hard plaque, aka calcium. That why cardiologist use the CAC score to assess artery health.

Bottom line, we want our LDL particles to be 'fluffy' and large, so they don't burrow down into the inner lining of the arteries which build up and eventually erupt causing blockage.

The standard lipid panel is outdated and while it is helpful, it doesn't give a full evaluation of LDL, HDLs, cholesterol...

Request advanced testing like the NMR LipoProfile (Nuclear Magnetic Resonance LipoProfile.

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However, recent research shows that lipoprotein particle number is more predictive of cardiovascular risk than cholesterol levels. The Nuclear Magnetic Resonance (NMR) LipoProfile test assesses the number of lipoprotein particles, sizes of lipoproteins, levels of cholesterol, and patient risk categories. Furthermore, it enables the identification of patients with underestimated cardiovascular risks—those with a discordant high number of low-density lipoprotein (LDL) particles (LDL-P) despite low cholesterol levels.

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LINK