More CWD in Ms...... Marshal county.

Looks like a good chance Tennessee has it due to proximity. Genie is out of the bottle now.

shite!!!!

Some where in between

If it does what they say it does, don’t you think they’d have more folks with that brain disease? It seems that cwd has been around a long time. I don’t like it, but I don’t care enough to quit eating deer.

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it does what they say it does, don’t you think they’d have more folks with that brain disease? It seems that cwd has been around a long time. I don’t like it, but I don’t care enough to quit eating deer.

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From what I understand the only time a human is at risk of contracting the disease is if you eat parts of the nervous system. Specifically the brain.

I don't know many baws frying up the frontal cortex and I think the backstrap is fine

From some folks I know in Mississippi, it is spread through deer slaver.

Mother F.

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Doesn't appear that ANY coyotes or Coons have been shown to be susceptible to the disease. That gives me confidence that this is not a threat to humans as well.

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do you know of any coyotes or coons tested?

I would do a bit of research before becoming so dismissive here are a few things to think about.

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In 2001, the case of a 25-year-old man who reportedly died of a prion disease after an illness lasting ˜22 months was investigated (Table 2). Although this man had hunted deer only rarely, his grandfather hunted deer and elk throughout much of the 1980s and 1990s and regularly shared the venison with the case-patient’s family. The grandfather primarily hunted in southeastern Wyoming, around the known CWD-endemic area. The case-patient’s illness began with a seizure and progressed to fatigue, poor concentration, and depression. Memory loss, ataxia, speech abnormalities, combative behavior, and recurrent seizures also developed. Histopathologic, immunohistochemical, and Western blot testing of brain autopsy samples confirmed a prion disease diagnosis. Analysis of the prion protein gene indicated a P102L mutation coupled with valine at the polymorphic codon 129 in the mutant allele, confirming a diagnosis of Gerstmann-Sträussler-Scheinker syndrome (GSS). This case-patient was unusually young even for a person with a GSS P102L mutation. It remains unknown whether the possible exposure of the case-patient to CWD-infected venison potentially contributed to the early onset of his prion disease.

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One of the patients was a 61-year-old woman who grew up in an area where this disease is known to be endemic, and she ate venison harvested locally. She died in 2000, and analysis of autopsy brain specimens confirmed that the patient’s CJD phenotype fit the MM1 subtype, with no atypical neuropathologic features. The second patient was a 66-year-old man who was reported to have eaten venison from two deer harvested in a CWD-endemic area. Both deer tested negative for CWD, and the patient’s illness was consistent with the MM1 CJD phenotype.

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Well that’s scary. Are you on the “I quit eating deer” side?