No such thing as 'fat but fit', major study finds

Print out copies of this study and hand it to any fat frick who tries to claim otherwise.

We need to kill this "obese is ok" mindset with extreme prejudice.

Anything over 16% body fat is fat whether you like it or not. Anything over 20% is overweight and anything over 25% is obese

Not really, Charles Atlas. I'm not calling a 17% person fat......a tad over weight? Sure.

So you can't lift much with your wiry girl frame? Ladies see that as weakness. You projecting weakness bro?

I work out with a guy named Larry. He weighs 330 pounds and he can jump higher than I can. Dude may be big or "fat", but he has unreal strength and athleticism.

He's definitely "fit" imo.

And looking good doesn't mean you're very fit. I see guys who are shredded and look good but they are weak with poor coordination/balance.

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coordination/balance.

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What does this have to do with being "fit?"

There's a guy that will swim at one of the pools I swim at who is an easy 330 lbs. But the dude is a pretty decent swimmer. There are certainly people faster than him, but he's easily above average. Swims relatively fast, and puts in some pretty long sessions. To do that, you have to be at least remotely aerobically "fit". Now he definitely has a caloric intake issue. So he's not in good shape. But it's hard to say that he isn't, at least in some way, "fit".

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Body fat % has to do with being fit.

What do coordination and balance have to do with being fit?

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Be careful, this thread will nosedive into a Crossfit thread if you aren't careful.

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People who are obese run an increased risk of heart failure

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Water runs increased chance of being wet

The proper way to view fat/adiposity is as a nefarious functioning endocrine organ

At its roots, the underlying problems are:

-inflammation
-hormonal disruption









NIH

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How might obesity increase the risk of cancer?
Several possible mechanisms have been suggested to explain how obesity might increase the risks of some cancers.

Obese people often have chronic low-level inflammation, which can, over time, cause DNA damage that leads to cancer. Overweight and obese individuals are more likely than normal-weight individuals to have conditions or disorders that are linked to or that cause chronic local inflammation and that are risk factors for certain cancers (26). For example, chronic local inflammation induced by gastroesophageal reflux disease or Barrett esophagus is a likely cause of esophageal adenocarcinoma. Obesity is a risk factor for gallstones, a condition characterized by chronic gallbladder inflammation, and a history of gallstones is a strong risk factor for gallbladder cancer (27). Chronic ulcerative colitis (a chronic inflammatory condition) and hepatitis (a disease of the liver causing inflammation) are risk factors for different types of liver cancer (28).

Fat tissue (also called adipose tissue) produces excess amounts of estrogen, high levels of which have been associated with increased risks of breast, endometrial, ovarian, and some other cancers.

Obese people often have increased blood levels of insulin and insulin-like growth factor-1 (IGF-1). (This condition, known as hyperinsulinemia or insulin resistance, precedes the development of type 2 diabetes.) High levels of insulin and IGF-1 may promote the development of colon, kidney, prostate, and endometrial cancers (29).

Fat cells produce adipokines, hormones that may stimulate or inhibit cell growth. For example, the level of an adipokine called leptin, which seems to promote cell proliferation, in the blood increases with increasing body fat. And another adipokine, adiponectin—which is less abundant in obese people than in those of normal weight—may have antiproliferative effects.

Fat cells may also have direct and indirect effects on other cell growth regulators, including mammalian target of rapamycin (mTOR) and AMP-activated protein kinase.

Other possible mechanisms by which obesity could affect cancer risk include changes in the mechanical properties of the scaffolding that surrounds breast cells (30) and altered immune responses, effects on the nuclear factor kappa beta system, and oxidative stress

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Using polarity, skeletal muscle and bone density can/should be viewed as opposing forces that combat the issues above..

Goals:
maximize muscle mass, bone density through load bearing / resistance exercise (minimal effective dose principle)

more muscle = enhanced glucose partitioning = increased insulin sensitivity = decreased insulin resistance

I have commented ad nauseam here in the past about proper nutrition

When viewed through this lens (instead of a strictly thermodynamic math problem), much becomes clearer....

Dynamic inputs into a dynamic system